Take NMN to Boost NAD+ Levels

Currently, one of the most promising anti-aging targets being investigated is an important molecule called nicotinamide adenine dinucleotide (NAD+). NAD+ mediates the production of the energy cells need to function and survive, fueling key enzymes that repair DNA damage. This essential molecule decreases with age.

Many scientists hypothesize that age-related declines in NAD+ underlie the organ decline that characterizes aging. So by restoring NAD+, our cells become healthier, our organs become healthier, and we become healthier. If this can be achieved, the reduction in high-mortality age-related diseases such as cardiovascular disease, neurodegenerative disease and cancer will allow us to live longer.

How can we restore our NAD+ levels? Since NAD+ is produced naturally, our cells have the machinery needed to make NAD+ on their own. Our cells build molecules like a factory assembly line, where each component is a precursor to the next. The biochemical precursor to NAD+ is called Nicotinamide Mononucleotide (NMN). Unlike NAD+ itself, NMN can be taken orally, so it can be taken in supplement form to boost NAD+ levels.

NMN is used in the production of NAD+, which provides the energy our cells need to function. Several factors are thought to underlie the aging process, a lack of cellular energy being one of them. Genetic instability due to DNA damage is also one of these factors. NAD+ plays a key role in activating enzymes that maintain DNA integrity, thereby promoting genetic stability. Given its central role in these cellular processes, the potential benefits of using NMN to boost NAD+ extend to virtually all body systems.

As our blood vessels become dysfunctional as we age, blood flow to the brain can be impaired, leading to cognitive impairment. NMN has been shown to increase blood flow to the brain and improve cognitive function in mice. We can have a stroke when a blood vessel in our brain becomes clogged, which damages our brain tissue. NMN has been shown to not only delay stroke onset but also prevent stroke damage and improve cognitive and mitochondrial health after stroke in rodents.

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