The relationship between β-Nicotinamide Mononucleotide and body aging

The theory of cell aging points out that the body's aging is mainly due to the decline of cell function,β-Nicotinamide Mononucleotide and the decline of cell function is largely caused by the dysfunction of mitochondria. Mitochondria are the energy factories of the body. When mitochondria are damaged,β-Nicotinamide Mononucleotide excessive reactive oxygen species (ie, oxidative free radicals) are generated.

Reactive oxygen causes oxidative damage to mitochondrial proteins, lipids and nucleic acids,β-Nicotinamide Mononucleotide thereby further inhibiting oxidative phosphorylation and aggravating the production of reactive oxygen species,β-Nicotinamide Mononucleotide making cells unable to produce enough energy and eventually leading to apoptosis. This is the reason why people will slowly age. Studies have shown that the level of nad+ is particularly important during the entire process of energy production and damage. nad+ full name nicotinamide adenine dinucleotide,β-Nicotinamide Mononucleotide also known as coenzyme i or noga factor,β-Nicotinamide Mononucleotide participates in thousands of redox enzyme reactions in the human body.

With the increase of age,β-Nicotinamide Mononucleotide the level of nad+ (nicotinamide adenine dinucleotide, an important coenzyme for cell energy conversion) in the human body is gradually decreasing,β-Nicotinamide Mononucleotide resulting in a decrease in mitochondrial activity and accelerating the senescence of mitochondria, cells and the entire body. Enter a vicious circle. At this time,β-Nicotinamide Mononucleotide it needs to be supplemented from an external source.

β-nicotinamide mononucleotide (NMN) can directly participate in the synthesis of nad+ in the cell. Therefore,β-Nicotinamide Mononucleotide supplementation of nmn can effectively delay aging,β-Nicotinamide Mononucleotide and is expected to prevent and treat aging-related diseases.

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